ID 369 - Miedź

PL: Miedź
EN: Copper
Pdf: copper

Oświadczenie (2)

1. Charakterystyka żywności / składnika

The food constituent that is the subject of the health claim is copper which is a well recognised nutrient and is measurable in foods by established methods.
Copper occurs naturally in foods and is authorised for addition to foods (Annex I of the Regulation (EC) No 1925/20064 and Annex I of Directive 2002/46/EC5). This evaluation applies to copper naturally present in foods and those forms authorised for addition to foods (Annex II of the Regulation (EC) No 1925/2006 and Annex II of Directive 2002/46/EC).
The Panel considers that the food constituent, copper, which is the subject of the health claim, is sufficiently characterised.

2.8. Metabolizm cholesterolu (ID 369)

The claimed effect is “cholesterol and glucose metabolism”. The Panel assumes that the target population is the general population.
The Panel considers that normal cholesterol metabolism is beneficial to human health.

2.9. Metabolizm glukozy (ID 369)

The claimed effect is “cholesterol and glucose metabolism”. The Panel assumes that the target population is the general population.
The Panel considers that normal glucose metabolism is beneficial to human health.

3. Naukowe uzasadnienia wpływu na zdrowie człowieka

Copper is a component of some enzymes, cofactors, and proteins in the body. Among those copper metalloenzymes that have been identified in humans are: monoamine oxidase, diamine oxidase, lysyl oxidase, peptidylglycine-α-amidating monooxygenase, caeruloplasmin, ferrooxidase II, cytohrome c oxidase, dopamine β-hydroxylase, copper/zinc superoxide dismutase, and tyrosinase. The biochemical role of copper is primarily catalytic, with many copper metalloenzymes acting as oxidases to achieve the reduction of molecular oxygen (Biesalski et al., 1997; Garrow et al., 2000; EVM, 2002; Gibney et al., 2002; Mann and Truswell, 2001; Sadler et al., 1999; Wenzel, 1999).

3.8. Metabolizm cholesterolu (ID 369)

A textbook and three consensus opinions/reports from authoritative bodies were cited to substantiate the claimed effect.
The role of copper on cholesterol metabolism is not well understood nor well established in humans (Turnlund, 2006). Blood cholesterol increases in animals fed copper-deficient diets, but the effects of
either copper deficiency or copper supplementation in humans are not consistent (Strain, 1994; Medeiros and Wildman, 1997, JHCI, 2003).
The Panel concludes that a cause and effect relationship has not been established between dietary intake of copper and normal cholesterol metabolism in humans.

3.9. Metabolizm glukozy (ID 369)

A textbook and three consensus opinions/reports from authoritative bodies were cited to substantiate the claimed effect.
The role of copper in the regulation of glucose metabolisms in not well understood nor well established in humans (Turnlund, 2006; JHCI, 2003)
The Panel concludes that a cause and effect relationship has not been established between dietary intake of copper and normal glucose metabolism in humans.

5. Warunki i możliwe ograniczenia stosowania oświadczenia

The Panel considers that in order to bear the claim a food should be at least a source of copper as per Annex to Regulation (EC) 1924/2006. Such amounts can be easily consumed as part of a balanced diet. Tolerable Upper Intake Level (UL) has been established for copper as 5 mg/day in adults and
during pregnancy and lactation. For children and adolescents UL was established as 1 mg/day for 1-3 years, 2 mg/day for 4-6 years, 3 mg/day for 7-10 years, 4 mg/day for 11-17 years (SCF, 2003).

Warunki i możliwe ograniczenia stosowania oświadczenia

See above