ID 356 -
Jod
PL: Jod
EN: Iodine
Pdf: iodine
Oświadczenie (2)
- funkcji wzroku
- zdrowie oczu
1. Charakterystyka żywności / składnika
The food constituent that is the subject of the health claims is iodine, which is a well recognised nutrient and is measurable in foods by established methods.
Iodine occurs naturally in foods and is authorised for addition to foods (Annex I of the Regulation (EC) No 1925/20064 and Annex I of Directive 2002/46/EC5). This evaluation applies to iodine naturally present in foods and those forms authorised for addition to foods (Annex II of the Regulation (EC) No 1925/2006 and Annex II of Directive 2002/46/EC).
The Panel considers that the food constituent, iodine, which is the subject of the health claims, is sufficiently characterised.
2.3. Utrzymanie prawidłowego wzroku (ID 356)
The claimed effects are “eye health” and “visual function”. The Panel assumes that the target population is the general population.
“Eye health” has not been defined in the list. From the proposed wordings the Panel assumes that the claimed effect relates to maintenance of normal vision.
The Panel considers that maintenance of normal vision is beneficial to human health.
3. Naukowe uzasadnienia wpływu na zdrowie człowieka -
Iodine is an essential constituent of the thyroid hormones, which have important modifying or permissive roles in growth and development and there is a large body of evidence indicating a crucial role for iodine in growth and development (Sadler et al., 1999; IoM, 2002; Garrow et al., 2000; Strain and Cashman, 2002). A wide spectrum of iodine deficiency disorders (IDD) have been observed, depending on the degree of deficiency and the life stage at which the deficiency occurs. These disorders range from mild goitre to the most severe forms of endemic cretinism (congenital, severe, irreversible mental and growth retardation). Other symptoms of severe IDD (arising from iodine deficiency in the foetus) include deaf-mutism, squint, disorders of stance and gait, and dry skin and swollen subcutaneous tissue (Delange, 2000; EVM, 2003; WHO, 2007).
3.3. Utrzymanie prawidłowego wzroku (ID 356)
One reference was cited to substantiate the claim effect. The reference was a study on distribution of iodine in a porcine eye model following iontophoresis (Rieger et al., 1995).
The Panel notes that squint and eye problems result from severe foetal iodine deficiency rather than from IDD onset in adulthood.
The Panel notes that iodine has no specific role in the eye.
The Panel concludes that a cause and effect relationship has not been established between the dietary intake of iodine and maintenance of normal vision.
5. Warunki i możliwe ograniczenia stosowania oświadczenia
The Panel considers that in order to bear the claims a food should be at least a source of iodine as per Annex to Regulation 1924/2006. A Tolerable Upper Intake Level (UL) has been established for iodine as 600 μg/day in adults and during pregnancy and lactation. For children and adolescents UL was established as 200 μg/day for 1-3 years, 250 μg/day for 4-6 years, 300 μg/day for 7-10 years, 450 μg/day for 11-14 years and 500 μg/day for 15-17 years (SCF, 2002). The target population is the general population.
Warunki i możliwe ograniczenia stosowania oświadczenia
MINIMUM 15% RDA