ID 266 -
Miedź
PL: Miedź
EN: Copper
Pdf: copper
Oświadczenie (4)
- Miedź przyczynia się prawidłowego pozyskiwania energii w procesach metabolicznych
1. Charakterystyka żywności / składnika
The food constituent that is the subject of the health claim is copper which is a well recognised nutrient and is measurable in foods by established methods.
Copper occurs naturally in foods and is authorised for addition to foods (Annex I of the Regulation (EC) No 1925/20064 and Annex I of Directive 2002/46/EC5). This evaluation applies to copper naturally present in foods and those forms authorised for addition to foods (Annex II of the Regulation (EC) No 1925/2006 and Annex II of Directive 2002/46/EC).
The Panel considers that the food constituent, copper, which is the subject of the health claim, is sufficiently characterised.
2.4. Metabolizm energetyczny (ID 266)
The claimed effect is “energy production”. The Panel assumes that the target population is the general population.
The Panel considers that normal energy-yielding metabolism is beneficial to human health.
3. Naukowe uzasadnienia wpływu na zdrowie człowieka
Copper is a component of some enzymes, cofactors, and proteins in the body. Among those copper metalloenzymes that have been identified in humans are: monoamine oxidase, diamine oxidase, lysyl oxidase, peptidylglycine-α-amidating monooxygenase, caeruloplasmin, ferrooxidase II, cytohrome c oxidase, dopamine β-hydroxylase, copper/zinc superoxide dismutase, and tyrosinase. The biochemical role of copper is primarily catalytic, with many copper metalloenzymes acting as oxidases to achieve the reduction of molecular oxygen (Biesalski et al., 1997; Garrow et al., 2000; EVM, 2002; Gibney et al., 2002; Mann and Truswell, 2001; Sadler et al., 1999; Wenzel, 1999).
3.4. Metabolizm energetyczny (ID 266)
Copper participates as a cofactor for specific enzymes and electron transport proteins involved in many oxidative reactions that break down fats in fat tissue to produce energy in the form of ATP.
The copper-dependent enzyme cytochrome c oxidase plays a critical role in cellular energy production. Copper deficiency results in a decrease of cytochrome c oxidase activity and the respiratory capacity of mitochondria, particularly in liver, heart, and brain (Linder, 1991).
The Panel concludes that a cause and effect relationship has been established between the dietary intake of copper and normal energy-yielding metabolism. However, the evidence provided does not establish that inadequate intake of copper leading to impaired energy yielding metabolism occurs in the general EU population.
4.4. Metabolizm energetyczny (ID 266)
The Panel considers that the following wording reflects the scientific evidence: “Copper contributes to normal energy yielding metabolism”.
5. Warunki i możliwe ograniczenia stosowania oświadczenia
The Panel considers that in order to bear the claim a food should be at least a source of copper as per Annex to Regulation (EC) 1924/2006. Such amounts can be easily consumed as part of a balanced diet. Tolerable Upper Intake Level (UL) has been established for copper as 5 mg/day in adults and
during pregnancy and lactation. For children and adolescents UL was established as 1 mg/day for 1-3 years, 2 mg/day for 4-6 years, 3 mg/day for 7-10 years, 4 mg/day for 11-17 years (SCF, 2003).
Warunki i możliwe ograniczenia stosowania oświadczenia
Must at least be a source of mineral/s as per annex to regulation 1924/2006